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Fig. 1 | Rice

Fig. 1

From: Metabolomic and Transcriptomic Changes Induced by Potassium Deficiency During Sarocladium oryzae Infection Reveal Insights into Rice Sheath Rot Disease Resistance

Fig. 1

Rice deficiency in K shows hypersensitivity to S. oryzae infection. a The typical symptoms of ShR disease and the development of ShR among different K nutrition conditions. Numbers in the picture represent K concentrations, Fo and Fv/Fm values; dpi denotes days post inoculation; the abbreviations of the treatments, K represent potassium, “+” and “−” denote with or without K; + I represent inoculation with S. oryzae. b Lesions lengths (n = 30 biologically independent tillers were selected for measurement, and five representative tillers were used for each replicate). c Volcano plots showing the up- and down-regulated genes in K-starved plants versus K-sufficient plants before inoculation. d Principal component analysis (PCA) of K-starved and K-sufficient FLS based on the corresponding metabolome, transcriptome, and ionome datasets before S. oryzae inoculation. The metabolite relative intensity values (n = 6 biological replicates), FPKM values for transcriptomes (n = 3 biological replicates), and elemental contents (n = 3 biological replicates) were used for PCA. e PCA of K-starved and K-sufficient FLS based on the corresponding metabolome, transcriptome, and ionome datasets at 5 days upon S. oryzae inoculation. Biological replicates for each treatment are outlined by the circles

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